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CLC number: R544.1

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Received: 2002-10-21

Revision Accepted: 2003-02-20

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Journal of Zhejiang University SCIENCE A 2003 Vol.4 No.5 P.620-622

http://doi.org/10.1631/jzus.2003.0620


Relationship between cholecystolithiasis and polypoid gallbladder


Author(s):  WU Ai-jiao, LI Ying-qi, DU Li-an

Affiliation(s):  Zhejiang University Hospital, 310027 Hangzhou, China; more

Corresponding email(s):   wuaijiang1231@sina.com

Key Words:  Polypoid gallbladder (PLG), Cholecystolithiasis, Atypital hyperplasia


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WU Ai-jiao, LI Ying-qi, DU Li-an. Relationship between cholecystolithiasis and polypoid gallbladder[J]. Journal of Zhejiang University Science A, 2003, 4(5): 620-622.

@article{title="Relationship between cholecystolithiasis and polypoid gallbladder",
author="WU Ai-jiao, LI Ying-qi, DU Li-an",
journal="Journal of Zhejiang University Science A",
volume="4",
number="5",
pages="620-622",
year="2003",
publisher="Zhejiang University Press & Springer",
doi="10.1631/jzus.2003.0620"
}

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%A WU Ai-jiao
%A LI Ying-qi
%A DU Li-an
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%N 5
%P 620-622
%@ 1869-1951
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%I Zhejiang University Press & Springer
%DOI 10.1631/jzus.2003.0620

TY - JOUR
T1 - Relationship between cholecystolithiasis and polypoid gallbladder
A1 - WU Ai-jiao
A1 - LI Ying-qi
A1 - DU Li-an
J0 - Journal of Zhejiang University Science A
VL - 4
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SP - 620
EP - 622
%@ 1869-1951
Y1 - 2003
PB - Zhejiang University Press & Springer
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DOI - 10.1631/jzus.2003.0620


Abstract: 
Objective: To study the relationship between cholecystolithiasis and polypoid gallbladder(PLG), 260 patients with polypoid gallbladder were investigated. The patients were divided into 2 groups: group A (PLG combined with cholecystolithiasis) and group B (without cholecystolithiasis). The clinical pathological characteristics were analyzed. The intestinal epithelium metaplasia and atypical hyperplasia of the gallbladder mucosa were observed under light microscope. Results: Intestinal epithelium metaplasia and atypical hyperplasia of gallbladder mucosa were found in 47 of the 260 cases. The pathological lesions included 16 gallbladder carcinoma, 11 adenomatosis polyp, 5 myoadenoma, 7 cholesterol polyp, 4 inflammatory polyp and 4 adenomatosis hyperplasia, which occurred in 26 and 21 patients in group A and group B, i.e. 44.0% and 10.3% respectively. The difference between group A and group B was statistically significant (P<0.01). Conclusion: cholecystolithiasis and the succeeding inflammatory reaction is a risk-factor for the polypoid gallbladder to develop tumour.

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Reference

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